Dodaj do ulubionych

cukier czy fruktoza? pyt. do Joggera

IP: *.pl 05.04.02, 08:53
Joggerze, pomóż proszę....
Mam problem ze słodzeniem herbaty. Zaczełam używać fruktozy, ale czy przy
odchudzaniu rzeczywiście jest lepsza od cukru? Nie cierpię chemicznego smaku
słodzików, zreszta przeczytałam w postach że aspartam nie jest taki genialny.
Nie moge się oduczyć słodzenia herbaty, czym mogę słodzić gdy chce schudnąć
kilka kilogramów i pozbyć sie części tłuszczyku na biodrach. Ćwiczę na
siłowni, widzę efekty wszędzie ale nie w rejonie bioder.

Obserwuj wątek
    • jogger Re: cukier czy fruktoza? pyt. do Joggera 05.04.02, 16:14
      O fruktozie czytałem, że potrafi sprytnie omijać enzymy trawiące, i w wątrobie
      jest bezpośrednio metabolizowana do postaci tłuszczu. To już chyba zwykły
      cukier jest lepszy. Ja przerzuciłem się już dawno na herbaty ziołowe, zieloną,
      yer-vitę itp. wynalazki, które niesłodzone smakują lepiej. Natomiast na lokalny
      tłuszczyk - tylko lokalne ćwiczenia przegrzewające, np. na biodra - na
      zakończenie sesji na siłowni 30 min twistera non-stop.
      • Gość: dan Re: cukier czy fruktoza? pyt. do Joggera IP: *.warszawa.cvx.ppp.tpnet.pl 05.04.02, 17:01
        Mam nadzieje, ze ponizszy artykul rozwieje wiele mitow wokol frukntozy. Autor-
        Lyle McDonald jest obecnie najbardziej cenionym przez kulturystow dietetykiem.

        FRUCTOSE
        by Lyle McDonald

        As with so many other aspects of nutrition, especially bodybuilding and sports
        nutrition, beliefs about the sugar fructose (more commonly known as fruit
        sugar) vary widely. On the one hand, because of its low glycemic index (GI) and
        general lack of insulin response, many people consider fructose an ideal sugar,
        that should be used to replace other sugars especially for diabetics (1). There
        is also some indication that fructose may blunt appetite and affect food choice
        (2,3). Finally, because of the low insulin response, it's been suggested that
        fructose before or during exercise might allow increased fat utilization during
        exercise, while still maintaining blood glucose levels (4).

        At the other end of the spectrum, because of differences in its metabolism
        compared to other sugars, and the known effect of fructose on blood
        triglyceride (fat) levels, many nutritional authorities (most notably John
        Parillo) consider fructose and fruit a sort of nutritional satan that will only
        make you fat and that should be eliminated from the diets of bodybuilders

        What Is Fructose, How Much Do We Get and
        Where Is It Found In the Diet?

        Fructose is one of three monosaccharides (single sugar molecules, the other two
        are glucose and galactose) that occurs naturally in foods. Sucrose (table
        sugar, a disaccharide) is also 1/2 fructose. However, free fructose only occurs
        naturally in a few foods, notably some fruits (hence its common name of 'fruit
        sugar') and honey with the majority of our dietary fructose coming from the
        ingestion of sucrose. Fructose is also found in small amounts in a few
        vegetables.

        In recent years, the amount of fructose being consumed has increased
        significantly. One large scale analysis puts average daily amounts in the range
        of 90-100 grams (8) which is a significant increase over the last 2 decades.
        This increase has been caused not only by an overall increase in the
        consumption of sugars, but also because of increased use of high fructose corn
        syrup (HFS). HFS contains can contain 42% or 55% free fructose, with the
        remainder being glucose. So, on top of an overall increase in fructose
        consumption, the use of HFS has caused a significant increase in the
        consumption of free fructose (8,9).

        Additionally, many health-food stores also sell crystalline fructose powder as
        a sweetener and both crystalline fructose and HFS are used in many commercially
        produced food products (10).

        Fructose Digestion and Metabolism

        During digestion in the stomach and small intestines, all dietary carbohydrates
        are eventually broken down to the monosaccharides glucose, fructose and
        galactose (found in milk in lactose). These are absorbed via specific
        transporters in the small intestine, bringing them into the portal vein, next
        stop the liver.

        While the small intestine seems to have an essentially unlimited (estimated
        around 5000 grams per day) capacity to absorb glucose and galactose, the
        absorption of fructose is a very different story. The consumption of as little
        as 35-50 grams of free fructose at once causes gastric upset, gas, bloating and
        diarrhea in a majority (60% or more) of people (11). However, the addition of
        glucose to the free fructose prevents the problem. And the ingestion of large
        amounts of sucrose (again, 1/2 fructose, 1/2 glucose) causes no such problem.
        It thus appears that our guts have evolved to absorb fructose only in the
        presence of other sugars and that large amounts of free fructose in the diet
        are non-physiological for humans. Since HFS also contains a significant amount
        of glucose, the malabsorption issue may not be a huge one for most processed
        foods. However, anyone considering using crystalline fructose as a sweetener
        should consider the potential problems with large amounts of free fructose. I
        should also mention that some individuals suffer from a hereditary fructose
        malabsorption syndrome (12), but this is typically identified at a very young
        age. Fructose free diets can be developed for these individuals.

        After digestion, fructose goes to the liver like all sugars, which is where the
        bulk of its metabolism occurs. This is also where fructose metabolism differs
        significantly from glucose metabolism, and is the source of much of the debate
        over the relative 'goodness' of fructose.

        Liver Metabolism of Sugars

        Although they share many intermediate steps in their metabolism, dietary
        glucose and fructose follow two distinct pathways in the liver. In fact, it has
        been known for quite some time that most dietary glucose goes straight through
        the liver with only minimal metabolism (13) while the majority of fructose is
        metabolized in the liver (14).

        This is part of the reason for the low glycemic index (GI) of fructose. Since
        it is metabolized almost exclusively in the liver, it has only a very small
        effect on blood glucose levels. Additionally, and also unlike glucose, the
        uptake of fructose into liver cells doesn't require insulin. Hence there is no
        need for the body to secrete insulin in response to dietary fructose ingestion.
        This is the basis for the claims that fructose is a superior carbohydrate
        source compared to glucose or sucrose (which recall is half glucose),
        especially for diabetics (1) who have poor control over blood glucose and
        insulin levels.

        At the same time, because of its extensive metabolism, and the pathways that it
        follows, in the liver, fructose can have negative effects on the overall
        metabolism of the body. So first let's compare and contrast the metabolism of
        glucose and fructose in the liver, and then look at some of the potential
        negatives of fructose (especially excess fructose) intake.

        But Just How Much Are We Talking About?

        A cursory examination of the research into fructose feedings in terms of the
        production of metabolites such as lactate, uric acid and especially VLDL
        triglycerides has shown distinctly varying results. For example, while some
        research has clearly shown an increase in VLDL triglyceride levels with
        fructose feedings (18-23), other research has not (24-28). Why the discrepancy?

        As with so many aspects of nutrition, it really comes down to two things: how
        much fructose they gave, and what population they gave it to. Let's get the
        population dynamic out of the way first. Studies have examined the effects of
        fructose in essentially 4 different groups: individuals with normal insulin and
        triglyceride levels, individuals with normal insulin but high triglyceride
        levels, individuals with high insulin and normal triglyceride levels, and
        people with high insulin and triglyceride levels. Different groups show a
        different susceptibility to the negative effects of fructose (and that still
        has to take amount into account).

        Overall, the last group, those with high insulin and high triglyceride levels
        tends to be the most sensitive to the negative effects of fructose in terms of
        increasing VLDL triglyceride levels (3). The other three groups show a
        distinctly less pronounced effect. This makes some sense as such individuals
        would normally have skewed physiology to begin with. I should point out that
        hyperinsulinemic/hypertriglyceridemic folks aren't very indicative of the
        average lean athlete consuming a healthy bodybuilding/sports oriented diet.

        But that brings us back to dose. Even in otherwise healthy individuals,
        fructose has been found to increase VLDL triglyceride levels so the potential
        for fat synthesis from fructose is apparently there. Again, looking at the
        studies as a whole, both negative and positive results are typically found.
        It's when you start looking at the
      • Gość: dan Re: cukier czy fruktoza? pyt. do Joggera IP: *.warszawa.cvx.ppp.tpnet.pl 05.04.02, 17:04
        It's when you start looking at the amounts given, that a pattern starts to
        develop. First, a select group of studies has used absurd and non-physiological
        amounts of fructose (200-500 grams per day, more than any human is probably
        capable of consuming under all but the most forced conditions) and invariably
        found increased triglyceride levels. Keep in mind that the average American
        diet only contains about 30-40 grams of fructose per day so we can pretty
        safely ignore those studies.

        Looking at the other studies which gave more reasonable amounts of fructose, as
        a recent review has done (29), we see a fairly standard pattern: at reasonable
        amounts of fructose (30-60 grams per day depending on the study), there is no
        negative effect on VLDL or triglyceride levels. At amounts higher than that (in
        the range of 80-90+ grams per day), there tend to be an increase in VLDL and
        triglyceride levels suggesting fat synthesis. This would tend to suggest a
        distinct cutoff point somewhere between those two values as an approximate
        maximum of fructose that can be consumed without causing significant
        triglyceride synthesis.

        In contrast, one study comparing 75 grams of fructose to 75 grams of glucose,
        found that, over 4 hours of study, while there was a small amount of de novo
        lipogenesis from the fructose, the net effect was that the body burned more fat
        than it produced (30). The fructose group also showed a higher thermic effect
        (meaning more calories were wasted as heat), most likely because of the high
        amount of metabolic processing that went on. However, and perhaps more
        importantly, despite very little fat synthesis in the fructose group, there was
        less fat burning in that same group. This occurred with an increased burning of
        carbohydrate in the fructose group.

        So it may be that, while fructose at moderate ((50 g/day or so) amounts doesn't
        increase fat synthesis per se significantly, it may slow fat loss by decreasing
        fat burning in the liver. That is, to a degree, the end result may be the same:
        whether the fructose is causing more fat synthesis, or less fat burning, the
        net effect on fat loss (which is determined by fat burning - fat intake) may be
        similar.

        As I mentioned above, the metabolic fate of fructose appears to depend on the
        metabolic state of the liver and the dieter. In the fasted state (as occurs
        while dieting), fructose will be used for energy and fat synthesis will be
        negligible, if it occurs at all. Fat burning may be decreased however. In the
        fed state (as occurs when not dieting), excess fructose can be converted to
        VLDL TG, increasing heart disease risk and bodyfat.

        Fructose Feeding and Exercise Performance

        Finishing up, I want to touch on the idea of fructose feeding during exercise.
        To my knowledge, no studies have examined the consumption of different types of
        carbohydrates prior to weight training, and all of the research done to date
        has been in endurance athletes. From a theoretical standpoint (including the
        low GI/insulin response), there are some good reasons to think that fructose
        feedings might be superior to glucose during endurance exercise. However, the
        research to date has not supported the theory. Fructose feeding before or
        during has been found to be either no better, or in fact worse in terms of
        performance of endurance exercise (3). As well, there is the issue of gastric
        upset with high amounts of fructose that limits how much can be given in the
        first place.

        As a final comment, there is the issue of post-workout carbohydrate and
        recovery. At this point, it should be no surprise to readers that the post-
        workout consumption of carbs and protein improves recovery, performance and
        protein synthesis (32,33). Studies examining different types of carbohydrate
        intake post workout have invariably found that glucose and glucose polymers
        refill muscle glycogen ideally, while fructose preferentially refills liver
        glycogen. In that refilling liver glycogen can be important from the standpoint
        of overall recovery and growth (the details are outside of the scope of this
        article), consuming a small amount of fructose (10% of the total carbohydrate
        content or roughly 10-20 grams) in the post-workout shake may be beneficial.
        But the majority of carbohydrates consumed should come from glucose and glucose
        polymers.

        Summing Up and Practical Recommendations

        Ok, an article like this wouldn't be worth much without some actual
        recommendations and real world application. First and foremost, it should be
        clear that in large enough amounts, fructose can certainly be detrimental both
        to health, by raising VLDL cholesterol and triglycerides and possibly to
        bodyfat levels for the same reason. However, at moderate intakes of fructose,
        in the range of 50-60 grams per day, fructose appears to pose little problem
        and certainly is not going to make or break a diet.

        For an athlete to avoid all sources of fructose, especially fruit, seems a bit
        silly and extreme (see below regarding pre-contest bodybuilding prep for a
        possible exception). However, there is probably a good reason to avoid high
        fructose corn syrup as much as is reasonably possible. Readers should realize
        that many sports food companies are using fructose and HFS in their products,
        so it's possible that athletes are being exposed to larger than normal amounts
        of fructose in their diets. Athletes and bodybuilders are encouraged to become
        avid label readers to see if HFS is listed as a primary ingredient.

        Individuals who are hyperinsulinemic or have high triglycerides to begin with
        may question whether using fructose in large amounts is beneficial and should
        consult with their doctor before making major nutritional changes. The majority
        of athletes, bodybuilders and otherwise healthy individuals are unlikely to
        have problems with either hyperinsulinemia or hypertriglyceridemia although it
        is a possibility.

        So, back to the ~50 g/day value. Noting that the average American diet may
        contain at least double that already, we might assume that athletes shouldn't
        be adding more fructose or fruit to their diet. But, we really have to ask
        whether or not that applies to bodybuilders and athletes, who typically avoid
        the commercial foods which most commonly containing fructose (meaning those
        containing high fructose corn syrup).

        Most bodybuilders and other athletes already avoid the majority of such foods
        and I would expect that their daily fructose intake is somewhat below the
        American average. Once again, note that many commercial products aimed at
        athletes, such as food bars and even some meal replacement powders, are
        increasing their use of fructose and HFS as a sweetener so it is possible that
        athletes are getting more fructose or HFS than they're aware of. How much is up
        to debate and speculation.

        So let's address the important question: What about fruit? Can it be part of a
        healthy bodybuilding/athletic diet, or should it be avoided as Parillo claims?
        To answer this we really need to look at the amount of fructose found in
        typical fruits.

        On average, fruits such as cherries, pears, bananas, grapes and apples contain
        anywhere from 5 to 7 grams of fructose in an average sized piece of fruit.
        Fruits such as strawberries, blueberries, oranges and grapefruit contain 2-3
        grams of fructose per 100 gram serving. Honey is an exception, containing 40
        grams of fructose per 100 gram serving, but its extreme sweetness would make
        eating a lot of it difficult. The point being that fruit is actually not a very
        large source of fructose in the first place. To get 50 grams of fructose per
        day from fruit alone would require an intake of approximately 10 pieces per
        day, far more than all but the most extreme intake would provide.


        So, summing up, like most aspects of bodybu
        • jogger Re: kazeina? pyt. do Dana 05.04.02, 17:59
          Super wyciąg, nie znam za bardzo McDonalda, czytywałem tylko właśnie w/w
          Parillo i Aceto. Czy nie znalazłbyś jeszcze opinii nt. kazeiny? Wg. mnie mleko
          ze względu na b. dużą zawartość kazeiny jest dobre do tuczu zwierząt, a nie dla
          ludzi. Dlatego dla 'uzależnionych' od mleka najlepsze byłoby humanizowane mleko
          w proszku dla dzieci:
          '
          Proteiny z mleka mieszają się z kwasem żołądkowym podczas trawienia i tworzą
          grudki z kazeiny i wapnia. Ponieważ mleko kobiece ma małą zawartość kazeiny,
          tworzy miękkie i łatwostrawne grudki w żołądku. Duża zawartość kazeiny w krowim
          mleku powoduje tworzenie się twardych i ciężkostrawnych grudek. Mleka w proszku
          oparte na białku serwatkowym są lekkostrawne, gdyż imitują proporcje protein w
          mleku kobiecym.
          '
          Stąd zresztą populaność białka z serwatki (whey protein).
          • Gość: dan Re: kazeina? pyt. do Dana IP: *.warszawa.cvx.ppp.tpnet.pl 14.04.02, 12:38
            No tak ale dzieki gorszej przyswajalnosci kazeina ma wieksze dzialanie
            antykataboliczne co jak sie okazuje ma w sumie istotniejsze znaczenie niz lepsze
            wlasciowosci anaboliczne serwatki i w konsekwenji skutkuje wyzszym bilansem
            azotowym.
            Z tym ze dla kulturystow lepsza jest serwatka bo przyjmuja ja czesto i w duzych
            ilosciach, co zapewnia rownie wysoki wskaźnik antykataboliczny, a dzieki lepszym
            wlasciwosciom anabolicznym daje to jeszcze wyzszy bilans niz w przypadku kazeiny.

            Wiecej szczegolow znajdziesz tutaj:
            ajpendo.physiology.org/cgi/content/full/280/2/E340
      • Gość: dan Re: cukier czy fruktoza? pyt. do Joggera IP: *.warszawa.cvx.ppp.tpnet.pl 05.04.02, 17:07
        So, summing up, like most aspects of bodybuilding and athletic nutrition, there
        are few absolutes. While there is no doubt that large amounts of fructose are
        both non-physiological and potentially harmful, it certainly appears that low
        to moderate amounts of fructose, and yes fruit, can be included in a
        bodybuilding or athletic diet. From the standpoint of liver glycogen and
        maintaining an anabolic state, small amounts of fructose probably should be
        included in the diet. Considering the other nutrients (fiber, vitamins and
        minerals) present in fruit, it seems silly to exclude them from the diet based
        on the rather small amount of fructose present

        1. Gerrits, PE and Tsalikian, E. Diabetes and fructose metabolism. Am J Clin
        Nutr (1993) 58 (suppl): 796s-799s.

        2. Moyer, AE and Rodin, J. Fructose and behavior: does fructose influence food
        intake and macronutrient selection. Am J Clin Nutr (1993) 58 (suppl): 810s-
        814s.

        3. Henry, RR and Crapo, PA. Current issues in fructose metabolism. Ann Rev Nutr
        (1991) 11: 21-39.

        4. Craig, BW. The influence of fructose feeding on physical performance. Am J
        Clin Nutr (1993) 58 (suppl): 815s-819s.

        4a. Web article at: www.parrillo.com/sng/tsb_det...946114869&id=45

        5. O'Dell, BL. Fructose and mineral metabolism. Am J Clin Nutr (1993) 58: 771s-
        778s.

        6. Dills, Jr. WL. Protein fructosylation: fructose and the Maillard reaction.
        Am J Clin Nutr (1993) 58 (suppl): 779s-787s.

        7. Glinsmann WH and Bowman, BA. The public health significant of dietary
        fructose. Am J Clin Nutr (1993) 58 (suppl): 820s-823s.

        8. www.ers.usda.gov:80/publicat...B965/sb965h.pdf Table 2.

        9. Vuilleumier, S. Worldwide production of high-fructose syrup and crystalline
        fructose. Am J Clin Nutr (1993) 58 (suppl): 733s-736s.

        10. Hanover, ML and White, JS. Manufacturing, composition, and applications of
        fructose. Am J Clin Nutr (1993) 58 (suppl): 724s-732s.

        11. Riby, JE et. al. Fructose absorption. Am J Clin Nutr (1993) 58 (suppl):
        748s-753s.

        12. Ali, M, et. al. Heriditary Fructose Intolerance. J Med Genet (1998) 35:353-
        365.

        13. McGarry JD et. al. From dietary glucose to liver glycogen: the full circle
        around. Ann Rev Nutr (1987) 7:51-73.

        14. Mayes, PA. Intermediate metabolism of fructose. Am J Clin Nutr (1993) 58
        (suppl): 754s-765s.

        15. De Jong M et. al. T4 uptake into the perfused rat liver and liver T4 uptake
        in humans are inhibited by fructose.Am J Physiol (1994) 266(5 Pt 1):E768-E775

        16. Hellerstein MK. Synthesis of fat in response to alteration in diet:
        insights from new stable isotope methodologies. Lipids (1996) 31 (suppl): S117-
        S125.

        17. Acheson KJ et. al. Glycogen storage capacity and de novo lipogenesis during
        massive carbohydrate overfeeding in man. Am J Clin Nutr (1988) 48: 240-247.

        18. Jeppesen, J et. al. Postprandial triglyceride and retinyl ester responses
        to oral fat: effects of fructose. Am J Clin Nutr (1995) 61: 787-791.

        19. Abraha, A et. al. Acute effect of fructose on postprandial lipaemia in
        diabetic and non-diabetic subjects. Br J Nutr (1998) 80: 169-175.

        20. Reiser, S et. al. Blood lipids, lipoproteins, apoproteins, and uric acid in
        men fed diets containing fructose or high-amylose cornstarch. Am J Clin Nutr
        (1989) 49: 832-839.

        21. Bantle, JP et. al. Metabolic effects of dietary fructose in diabetic
        subjects. Diabetes Care (1992) 15: 1468-1476.

        22. Bantle, JP et. al. Effect of dietary fructose on plasma lipids in healthy
        subjects. Am J Clin Nutr (2000) 72: 1128-1134.

        23. Swanson, JE et. al. Metabolic effects of dietary fructose in healthy
        subjects. Am J Clin Nutr (1992) 55: 851-856.

        24. McAteer EJ et. al. The effects of one month high fructose intake on plasma
        glucose and lipid levels in non-insulin-dependent diabetes. Diabetic Medicine
        (1987) 4: 62-64.

        25. Grigorscro, C et. al. Lack of detectable deleterious effects on metabolic
        control of daily fructose ingestion for 2 Mo in NIDDM patients. Diabetes Care
        (1988) 11: 546-550.

        26. Eunsook, TK et. al. Effects of fructose feeding on blood parameters and
        blood pressure in impaired glucose-tolerant subjects. J Am Diet Assoc (1988)
        88: 932-938.

        27. Koivisto VA and Yki-Jarvinen H. Fructose and insulin sensitivity in
        patients with type 2 diabetics. J Internal Med (1993) 233: 145-153.

        28. Malerbi, DA et. al. Metabolic effects of dietary sucrose and fructose in
        Type II diabetic subjects. Diabetes Care (1996) 19: 1249-1256.

        29. Hollenbeck, CB. Dietary fructose effects on lipoprotein metabolism and risk
        for coronary heart disease. Am J Clin Nutr (1993) 58 (suppl): 800s-809s.

        30. Tappy, L, et. al. Comparison of the thermogenic effect of fructose and
        glucose in normal humans. Am J Physiol (1986) 250: E718-E724.

        31. Tittelbach, TJ, et. al. Post-exercise substrate utilization after a high
        glucose vs. high fructose meal during negative energy balance in the obese.
        Obes Res (2000) 8: 496-505.

        32. Zawadzki et al. Carbohydrate-protein complex increases the rate of muscle
        glycogen storage after exercise. J Appl Physiol (1992) 72: 1854-1859.

        33. Rasmussen BB et. al. An oral essential amino acid-carbohydrate supplement
        enhances muscle protein anabolism after resistance exercise. J Appl Physiol.
        (2000) 88:386-92.




Nie masz jeszcze konta? Zarejestruj się


Nakarm Pajacyka